Viral Infections
Viral replication is detected by Pattern-recognition receptors (PRRs), which trigger an immune response, leading to the production of restriction factors, like Type 1 Interferons.
Directly-acting antivirals (DAAs) are very specific in targeting viral infections.
HSV and VSV
Herpes Simplex Virus (HSV) presents with painful blisters and ulcers on genitals/mouth
Most commonly due to HSV 1
It can reoccur and present with prodromal tingling

Varicella Zoster Virus (VZV) aka. Chickenpox, presents with a vesicular rash, accompanied by fever and fatigue, which dries up and crusts over within a week
It can re-activate later in life as Shingles - It remains latent in the spinal cord after the first infection, before reactivating along a dermatome - This presents with allodynia and hyperalgesia
N.B. Patients tend to develop Post-herpetic neuralgia (10% to 18%) once the rash resolves, in which they have persistent pain/burning in the affected area. Treatment is supportive, and most cases resolve within a few months. Early treatment with Aciclovir reduces the risk of this complication.
Both HSV and VZV are managed with Aciclovir or Valaciclovir. These drugs are insoluble in urine, so it can crystallise in the renal tubules when at a high dose, leading to renal failure.
CMV and EBV
Cytomegalovirus (CMV) is a major pathogen in solid organ and bone marrow transplant patients.
It can cause retinitis in immunocompromised patients
Epstein-Barr Virus (EBV) causes Infective Mononucleosis, aka. Glandular fever, which presents with a sore throat, macular rash, neck lymphadenopathy, fever, and malaise. It's diagnosed with the Monospot test.
Both CMV and EBV are managed with Ganciclovir or Valganciclovir.