Infective Endocarditis
Pathophysiology
Transient Bacteraemia – Can occur from poor dentition, dental infections, prosthetic valve, IVDU etc.
Damage to valve
Formation of vegetations – Endothelium is exposed, leading to thrombus formation around the central cluster of the pathogen = Vegetation
N.B. Immune complexes can be formed and deposited in organs like the kidneys, skin and eyes (Type 3 hypersensitivity reaction)
Risk factors:
Age
Male
Poor dentition and dental infections
IVDU
Co-morbid conditions - Prosthetic valves, Valvular HD, IV devices e.g. central lines, shunts, Haemodialysis, HIV
Causes:
Staph aureus (most common)
Strep viridans
Enterococci
Presentation
Main features that should make you immediately suspect IE are a Fever + New/changing heart murmur.
O/E - Tachycardia, Splinter haemorrhages, Osler nodes/Janeway lesions (very rarely seen)
N.B. Presentation is diverse and variable – Can present acutely and progress rapidly w/symptoms of HF (typically on normal valves), or it can present subacutely/chronically w/non-specific symptoms (typically on abnormal/prosthetic valves).
Investigations and Management
An ECG should be done straight away to rule out 1st degree heart block (Prolonged PR). If found, it's indicative of an Aortic root abscess, which is a rare, but severe, complication of IE. Managed with surgical repair.
Other investigations to do include:
Blood culture – 3 sets taken 30 mins apart from 3 separate peripheral sites
Bloods – FBC, CRP, U&E, LFT
Echo - 1st line imaging
Urine dip – Check for renal involvement
Other indications for surgical intervention include:
Extensive valve damage
Prosthetic valve endocarditis
Persistent infection despite medical therapy
Large vegetations
Serious embolisation
Fungal endocarditis
Progressive cardiac failure
Management is with long-term IV Abx.
Complications
Aortic root abscess (1st degree HB/Prolonged PR)
Acute valvular insufficiency → HF
Embolism e.g. stroke
Infection e.g. osteomyelitis, septic arthritis